ISSN 2149-2263 | E-ISSN 2149-2271
The Anatolian Journal of Cardiology - Anatol J Cardiol: 8 (2)
Volume: 8  Issue: 2 - April 2008
1.Co-editors participation, preparation of the new editorial board, where is the ethic? Our new affliction
Bilgin Timuralp
PMID: 18400626  Page 93
Abstract |Full Text PDF

2.Citric acid as a decalcifying agent for the excised calcified human heart valves
Necmi Köse, Barlas Naim Aytaçoğlu, Necat Yılmaz, H. Ali Döndaş, Lülüfer Tamer, Banu Coşkun, Özden Vezir, Nehir Sucu, Murat Dikmengil
PMID: 18400627  Pages 94 - 98
Objective: Cardiac valvular pathologies are frequently encountered as mechanical and functional disorders due to the calcification of the valves whatever the etiologies are. This pathophysiologic table usually ends up with valvular replacement. In this study we aimed to decrease/eliminate the calcium in the excised calcified human heart valves by using citric acid in vitro hence bringing about the question for possible oral treatment of calcification of the valves by citric acid ingestion. Methods: Fourteen pieces of mitral and/or aortic valves excised from 12 patients undergoing valve replacement were placed in a freshly prepared phosphate buffered saline solution containing 0.625% glutaraldehyde at +4 0C for 48 h. They were rinsed with 0.9% NaCl and divided into two groups; study and control. Control tissues were further treated in a freshly prepared solution with identical properties for another 5 days. Study tissues were placed into a solution containing 3.8% citric acid (pH 7.4) and kept for 48 h at +37 0C, then rinsed with 0.9% NaCl and transferred into a fresh solution containing 0.625% glutaraldehyde with phosphate buffer at 37 0C for 3 more days. Specimens were biochemically and histopathologically evaluated and compared using Mann Whitney U test. Results: Calcium and phosphate levels in the study group were lower than in the control group (852.5±913.41 µg g-1 vs 413.05±519.53 µg g-1, p=0.001 and 207.6±321.86 µg g-1 vs 124.4±289.48 µg g-1, p=0.035, respectively). Malondialdehyde and protein level values were changed insignificantly in the control and study groups. Histopathologic evaluation showed that collagen and elastin fibers were similar in both groups. In the study group, irregular and fusiform calcific formations around the collagen fibers were significantly decreased. Conclusions: Decalcifying human heart valves in vitro conditions with citric acid without an adverse change to the morphology of the valvular tissue specimens is meaningful. We believe that forwarding and looking for the answer to the question “whether systemic application of citric acid could lead to the decalcification and/or reduction of calcification in the native human heart valves” would be expressive.

3.Statin use is associated with decreased CD-40 ligand expression on T lymphocytes of coronary atheroma plaque in patients with stable coronary artery disease
Uğur Türk, Emin Alioğlu, İstemihan Tengiz, Ertuğrul Ercan, Reşat Mahmudov, Hamza Duygu, Cüneyt Türkoğlu
PMID: 18400628  Pages 99 - 103
Objective: Atherosclerosis is a chronic inflammatory disease. Statins suppress the inflammation in the plaque. This cross-sectional study was planned to evaluate the effect of statins on plaque T cell activation markers in patients with stable angina pectoris undergoing coronary intervention and atherectomy procedures. Methods: Twenty-six patients with stable angina with suitable for atherectomy coronary lesions were enrolled in the study. Fourteen of 26 patients who had been taking statin treatment for at least six months were assigned to the Group 1 (Statin group) and 12 patients who had not received any lipid lowering treatment comprised the Group 2 (Control group). Atherectomy specimens were studied with single and double immunohistochemical staining (CD25, CD69, and CD40L). Statistical analysis was performed using Student's t-test and Fisher's exact test. Results: There was no significant difference between the total tissue area of sections (Group 1: 8.4±0.9 mm2, Group 2: 7.8±0.9 mm2, p>0.05). CD3, CD25, CD69, and CD40L positive cells did not show statistically significant difference between the groups in unit area (mm2). There was no significant difference between the groups for percentage of T lymphocytes expressing CD25 (Group 1: 7.8±4.6%, Group 2: 7.8±5.9%, p=0.97) and CD 69 (Group 1: 12.9±4.6%, Group 2: 15.5±5.2%, p=0.203). The expression of CD40L was significantly lower in Group 1 than in Group 2 (Group 1: 4.8±3.9%, Group 2: 11.2±8.7%, p=0.034). Conclusion: We concluded that, statin treatment may decrease the expression of CD40L on plaque T lymphocytes in patients with stable angina pectoris.

4.Clonidine prevents development of hypertension in N (Omega)-Nitro-L-Arginine-treated rats
Süleyman Oktar, Selçuk İlhan, Hakkı Engin Aksulu
PMID: 18400629  Pages 104 - 110
Objective: Although there are some evidences on the contribution of increased sympathoadrenergic activity on long-term nitric oxide synthase (NOS) inhibition induced hypertension, the contribution of sympathetic activity to the development of this model of hypertension are not sufficiently studied. The aim of the present study is to investigate the effects of clonidine on blood pressure and vascular alpha-adrenergic receptors in the long-term N (omega)-nitro-L-arginine (L-NNA) treated rats. Methods: Sixty two Wistar rats were randomly divided into 8 groups. All groups were administrated L-NNA and/or clonidine in two different concentrations for ten days. L-NNA was administrated in concentrations of 15 and 45 mg/100ml to L-NNA15 and L-NNA45 groups, respectively. Clonidine was also administrated in concentrations of 150 and 225 µg/100ml to KLO150 and KLO225 groups, respectively. Blood pressure and heart rates were measured with tail-cuff method and plasma NOx levels with spectrophotometer. The a-adrenoreceptors responses were evaluated in thoracic aorta rings in “in vitro” conditions. Results: Clonidine prevented the L-NNA induced hypertension dose-dependently, but did not effect the heart rates decreased by L-NNA. The heart rates and blood pressure of normotensive rats were not changed by clonidine alone. Plasma NOx levels increased in L-NNA15 group (0.62±0.11 µmol/L, p=0.003) but did not change in other groups. The sensitivity of aorta to phenylephrine (-7.33±0.11 µmol/L, p=0.001) and clonidine (-7.60±0.27 µmol/L, p=0.003) in L-NNA45 group and phenylephrine (-6.94±0.13 µmol/L, p=0.002) in L-NNA15 group increased. The sensitivity of aorta to phenylephrine (7.93±0.16 µmol/L, p=0.001) in KLO225 group and to clonidine (-7.20±0.10 µmol/L, p=0.009) in KLO150 group increased. Conclusion: This study supports the idea suggesting that symphathetic nervous system activation is partly responsible for the development of the long-term NOS inhibition induced hypertension. In conclusion, it was shown for the first time that clonidine prevents the development of long-term NOS inhibition induced hypertension dose-dependently.

5.Increased lipoprotein(a) in metabolic syndrome: Is it a contributing factor to premature atherosclerosis?
Hüseyin Bozbaş, Aylin Yıldırır, Bahar Pirat, Serpil Eroğlu, Mehmet E. Korkmaz, İlyas Atar, Taner Ulus, Alp Aydınalp, Bülent Özin, Haldun Müderrisoğlu
PMID: 18400630  Pages 111 - 115
Objective: It is well known that patients with metabolic syndrome (MS) have a greater risk of developing coronary artery disease (CAD). However, the association of novel coronary risk factors with MS has not been well established. In this study, we sought to investigate the association of lipoprotein (a) [Lp(a)], homocysteine (Hcy), uric acid, and C-reactive protein (CRP) levels with MS. Methods: We enrolled 355 consecutive patients from our outpatient cardiology clinic into this cross-sectional, controlled study-186 with MS and 169 without MS, according to the Adult Treatment Panel III criteria. Serum Hcy, Lp(a), uric acid, and CRP levels were determined and compared between the groups. Results: The groups were homogenous with regard to age, sex, and other demographic variables (all p>0.05). As expected, the prevalence of hypertension (85.4% vs 55.6%, p<0.001) and dyslipidemia (78.3% vs 62.6%, p<0.05) were higher in patients in the MS group. Patients were comparable with respect to smoking (28.4% vs 24.8%, p =0.4) and family history of CAD (46.1% vs 40.8%, p=0.3). Patients with MS had significantly higher Lp(a) levels [29.2 (13.4-45.7) vs 16.2 (9.5-26.2) mg/dL; p<0.0001] compared with controls, whereas Hcy (12.2±4.8 vs 12.3±4.9 µmol/L; P=0.8), uric acid (5.7±1.6 vs 5.3±1.3 mg/dL; p=0.08), and CRP levels [6.0 (3.7-9.3) vs 5.1 (3.2-7.6) mg/L; p=0.07] were similar. Conclusion: Patients with MS seems to have increased serum levels of Lp(a), which might contribute to the premature atherosclerosis observed in these patients. Further research is needed to better clarify this issue.

6.Mentholated cigarette smoking induced alterations in left and right ventricular functions in chronic smokers
Özgür Çiftçi, Mustafa Çalışkan, Hakan Güllü, Aylin Yıldırır, Haldun Müderrisoğlu
PMID: 18400631  Pages 116 - 122
Objective: Possible acute effects of smoking mentholated cigarette on left and right ventricular function is not known. The aim of the study was to compare acute effects of normal and mentholated cigarettes smoking on both ventricular diastolic functions in chronic smokers. Methods: In a single-blinded, cross-over, open label and controlled study acute effect of smoking of the mentholated and the regular cigarettes was evaluated. Eighteen other than healthy regular cigarette smokers and 20 nonsmoker control subjects were included into the study. To compare the acute effects of mentholated and regular cigarette in each subject echocardiographic examination including tissue Doppler imaging (TDI) were performed at baseline, than in the smokers group TDI was measure 20-30 minutes after smoking two either cigarettes. Results: In response to smoking two cigarettes, mitral E/A values declined from 1.78±0.44 to 1.58±0.41 after the regular cigarette (p=0.0043) and from 1.78±0.44 to 1.53±0.40 after the mentholated cigarette (p=0.0035). Tricuspid E deceleration time values declined from 185.28±20.05 ms to 222.72±26.47 ms after the regular cigarette (p<0.001) and 185.28±20.05 ms to 241.53±47.63 ms after the mentholated cigarette (p<0.001). Smoking of mentholated cigarette, but not regular cigarette smoking, increased tricuspid E deceleration time and right ventricular isovolumic contraction time (p=0.044; p=0.024 respectively) and decreased the right ventricular Em values (p=0.027). Conclusion: Mentholated and regular cigarette smoking have acute detrimental effects on right and left ventricular systolic and diastolic function. Mentholated cigarettes cause additional unfavorable acute effects on especially right ventricular tissue Doppler velocities, relaxation and contraction indices compared to regular cigarettes.

7.Clinical and angiographic importance of right bundle branch block in the setting of acute anterior myocardial infarction
Uğur Arslan, Serhat Balcıoğlu, Yusuf Tavil, Murat Özdemir, Atiye Çengel
PMID: 18400632  Pages 123 - 127
Objective: To investigate functional status of patients (Killip class), left ventricular contractility, angiographic anatomy and severity of coronary lesions in patients with and without right bundle branch block (RBBB) in the setting of anterior myocardial infarction (MI). Methods: Patients who admitted to coronary care unit with the diagnosis of acute anterior MI between 1999 and 2005 were retrospectively searched from our database. Out of 792 patients, 37 had RBBB (RBBB group) either at admission or in the course of anterior MI. Forty patients who developed no intraventricular conduction defect during the course of anterior MI with the same demographic characteristics were selected as the control group. Results: Out of 37 patients, 30 had RBBB on admission and 7 developed RBBB in the course of acute MI. Left anterior descending artery (LAD) proximal lesion was more commonly detected in the RBBB group [23 (62.2%) vs. 11 (27.5%) patients, p=0.003]. Left ventricular ejection fraction was lower (33.0±4.2% vs 36.7±4.9%, p=0.003) and end-systolic volume was higher (84.1±24.9 ml vs 74.6±22.0 ml, p=0.012) in patients with RBBB. Number of patients with high Killip grade (III and IV) was more in the RBBB group [7 (18.9%) vs 3 (7.5%), RR: 1.75, %95 CI 0.92-3.32, p=0.14], and number of patients with Killip grade I was more in the control group [34 (85.0%) vs 22 (59.5%), p=0.012]. Besides mean Killip class was higher in the RBBB group (1.65±0.90 vs 1.25±0.67, p=0.03). Three patients (8.1%) in the RBBB group and 2 patients (5.0%) in the control group died during hospitalization (p=0.67). Conclusion: Left ventricular ejection fraction decreases and Killip grade increases in case of RBBB in the setting of acute anterior MI. Culprit lesion in patients with RBBB is more commonly a LAD proximal lesion and threatened myocardial tissue is larger in patients with RBBB.

8.The diagnostic value of multislice computed tomography in evaluation of coronary artery disease in patients with left bundle branch block
Mehmet Selim Nural, Mustafa Yazıcı, Muzaffer Elmalı, Sabri Demircan, Kamil Göl
PMID: 18400633  Pages 128 - 133
Objective: Noninvasive diagnostic methods do not supply sufficient data for patients with left bundle branch block (LBBB) accompanied with coronary artery diseases (CAD). Therefore, generally coronary angiography is required for these patients. Our aim was to evaluate the diagnostic value of multislice spiral computed tomographic (MSCT) coronary angiography to detect CAD in patients with LBBB. Methods: Sixty one patients (31 males, 30 females, mean age: 56±13 years) with LBBB who have determined stenosis higher than 50% in quantitative coronary angiography were included in the cross-sectional study. The MSCT coronary angiography was applied to the patients with a 16-detector MSCT scanner that has an electrocardiographic synchronization unit. Each coronary artery was evaluated segmentally in the images acquired from MSCT coronary angiographies and any detected stenosis higher than 50% was recorded. Results: The data of 793 coronary artery segments achieved from MSCT coronary angiographies of 61 patients (13 segments for each patient) were compared with the results of conventional coronary angiographies of the same patients. When all the segments evaluated were included in this comparison, the diagnostic accuracy, sensitivity, specificity, positive and negative predictive values of MSCT coronary angiography to detect stenosis higher than 50% were 91%, 67%, 97%, 85% and 92%, respectively. Also, it was observed that, MSCT coronary angiography has 80% sensitivity and 90% specificity to detect, at least, one segment coronary artery stenosis. Conclusion: The MSCT coronary angiography can be utilized as a noninvasive diagnostic method for patients with LBBB, in order to evaluate coronary artery disease.

9.Endovascular treatment of aortic lesions using the Medtronic ®Talent system: single center experience with mid-term follow-up
Barbaros E. Çil, Murat Canyiğit, Turan Türkmen Çiftçi, Bora Peynircioğlu, Tuncay Hazırolan, A. Gülsün Pamuk, Bora Farsak, Mustafa Yılmaz, Cem Yorgancıoğlu, Rıza Doğan, Metin Demircin, İlhan Paşaoğlu, Ferhun Balkancı
PMID: 18400634  Pages 134 - 138
Objective: The aim of this retrospective study is to investigate the safety and efficacy of endovascular repair of aortic lesions with the Medtronic ®Talent stent-graft system and to present mid-term results of endovascular aortic repair performed in our center. Methods: Between December 2002 and March 2007, 54 patients (6 women) with aortic (14 thoracic and 40 abdominal) lesions underwent treatment with ®Talent stent-graft. The average age of the patients was 64.8 (20-88) years. Duration of follow-up period ranged from 1 to 49 months (average 21 months). Indications for endovascular repair were degenerative aneurysm in 45, degenerative aneurysm and penetrating ulcer in 2, only penetrating ulcer in 1, traumatic thoracic isthmic transsection in 4 and vasculitic aneurysm secondary to Behcet’s disease in 2 patients. Results: Repair was performed with the tubular (16), aortouniiliac (1) or bifurcated (37) stent-grafts. Technical success rate was 100%. No death, major complication or need of immediate conversion to open repair was seen. Endoleak rate was 18.5% at 1 month follow-up period. Thirty-day mortality was 1.8% and morbidity (other than endoleaks) rate was 12.9%. During the follow-up period, secondary intervention was required in 12.9% of patients. Iliac limb occlusion was detected in 1 patient (1.8%). Graft migration causing type 1 endoleak and requiring open surgical treatment was seen in 1 patient (1.8%). Four patients (7.4%) are still under follow-up for type-2 endoleaks that do not require intervention. No graft infection or death due to aneurysm rupture was detected. Conclusion: Endovascular treatment of aortic lesions in selected patients with comorbid conditions using the ®Talent stent-graft exhibits a high degree of technical success with a low perioperative morbidity and mortality rate. The major disadvantage of endovascular aortic repair is necessity of life-long imaging follow-up and secondary interventions.

10.Human soluble tumor necrosis factor receptor I (sTNF-RI) and interleukin-I receptor antagonist (IL-I Ra) in different stages of acute rheumatic fever
Necil Kütükçüler, Neslihan E. Karaca, Betül Y. Sözeri, Güldane Koturoğlu, Zafer Kurugöl, Ruhi A. Özyürek, Güzide Aksu
PMID: 18400635  Pages 139 - 142
Objective: Acute rheumatic fever (ARF) results from an autoimmune response to infection with group A streptococci. Serum concentrations of two anti-inflammatory cytokines, interleukin-I receptor antagonist (IL-IRa) and human soluble tumor necrosis factor receptor I (sTNF-RI) were determined in patients with ARF at the time of admission and 3 months after treatment in order to evaluate changes in cytokine concentrations occurring during different stages of the disease. Methods: Serum concentrations of two anti-inflammatory cytokines, IL-I Ra and sTNF-RI, were investigated in children with ARF at the time of admission (n=21) and after 3 months following the cessation of treatment (n=15). The sTNF-RI and sIL-IRa were measured quantitatively in serum using enzyme-linked immunosorbent assay (ELISA). Results: Levels of IL-1Ra and sTNF-RI were found to be significantly higher during acute phase and remission period of ARF when compared to age-matched healthy controls (p=0.001 and p=0.0001, respectively). Conclusion: Our study demonstrated that two anti-inflammatory cytokines, serum sTNFRI and IL-1Ra, are increased in acute and remission stages of ARF reflecting activation of the cellular immune response. We suggest this increase might probably be generated in an effort to counteract the already increased concentrations of proinflammatory cytokines.

11.Myocardial performance index
Abdullah Uluçay, Ersan Tatlı
PMID: 18400636  Pages 143 - 148
Myocardial performance index (MPI) is a numeric value, which could be obtained by using cardiac time intervals. This numeric value is defined as the sum of isovolumetric contraction time (ICT) and isovolumetric relaxation time (IRT) divided by ejection time (ET) and could be calculated for each ventricle individually. Myocardial performance index has been invented as a Doppler index of combined systolic and diastolic myocardial performance in patients with primary myocardial systolic dysfunction. Myocardial performance index has prognostic value in various clinical settings seems to be independent of heart rate. In this review, clinical applications of MPI are scrutinized.

12.Global confusion on the diagnostic criteria for metabolic syndrome: What is the point that guidelines can not agree?
Selçuk Dağdelen, Tolga Yıldırım, Tomris Erbaş
PMID: 18400637  Pages 149 - 153
Several international and national associations have proposed their own diagnostic criteria for metabolic syndrome. Regarding the heterogeneity of these guidelines, some authors refused the usage of metabolic syndrome definition. Here we aimed to analyze the differences between the diagnostic criteria proposed for metabolic syndrome. We reviewed and compared these different guidelines’ criteria. We conclude that, despite the heterogeneity, there is a conceptual agreement on the definition of metabolic syndrome. But cut-off values and selection of diagnostic parameters are still conflicting. Diagnostic or definitive components of the syndrome (i.e., obesity, insulin resistance) should be considered separately from the associated-conditions (i.e., polycystic ovary, obstructive sleep apnea, microalbuminuria, non-alcoholic steatohepatitis etc.) of metabolic syndrome, during the course of diagnosis. Maintenance of the metabolic syndrome as a diagnostic category would still seem to be useful for an effective multiple cardiovascular risk prediction.

13.Thromboelastography and its use in cardiac surgery
Koray Ak, Nazan Atalan, Atike Tekeli, Selim İşbir, Ali Civelek, Nesrin Emekli
PMID: 18400638  Pages 154 - 162
Thromboelastography is an alternative method to conventional coagulation tests for the general evaluation of hemostatic system. Cardiac surgery with cardiopulmonary bypass is accomplished by complex alterations of hemostasis, including acquired dysfunction of platelets, consumption coagulopathy and increased fibrinolysis. Despite major advances in blood conservation methods and perioperative care of the patients, transfusion rates in cardiac surgery remain high. Thromboelastography has an ability to assess almost all components of haemostatic system globally. Currently, thromboelastography is used with standard coagulation tests to decrease the microvascular bleeding and homologous blood transfusion in cardiac surgery with cardiopulmonary bypass. In this review, we aimed to discuss thromboelastography technology and its usage in cardiac surgery.

14.Implantation of a cardioverter defibrillator on the side of a hemodialysis fistula to prevent loss of the alternative arterio-venous access
Ali Erdoğan, Monika Lüdemann, Harald Tillmanns, Till Spiegl
PMID: 18400639  Pages 163 - 164
Abstract |Full Text PDF

15.Persistent atrial fibrillation associated with gastroesophageal reflux accompanied by hiatal hernia
Hamza Duygu, Filiz Özerkan, Serkan Saygı, Serdar Akyüz
PMID: 18400640  Pages 164 - 165
Abstract |Full Text PDF

16.A complex congenital cardiovascular anomaly: persistence of left inferior and superior caval veins in conjunction with atrial and ventricular septal defects
Abdullah Doğan, Yasin Türker, Mehmet Özaydın, Nurullah Tüzün
PMID: 18400641  Pages 166 - 168
Abstract |Full Text PDF

17.Tricuspid valve mass: magnetic resonance imaging findings
Demet Erciyes, Cihan Duran, Mustafa Şirvancı, Murat Gülbaran
PMID: 18400642  Pages 168 - 169
Abstract |Full Text PDF

18.Midterm survival following repair of a giant left ventricular true aneurysm ruptured during operation and associated with papillary muscle rupture
Mehmet Çakıcı, Bahadır İnan, Sadık Eryılmaz, Mustafa Şırlak, Ümit Özyurda
PMID: 18400643  Pages 169 - 171
Abstract |Full Text PDF

19.Implantation of pulmonary valve without use of cardiopulmonary bypass: an innovative method
Süleyman Özkan, Salih Özçobanoğlu, Tankut Akay, Utku Alemdaroğlu, Sait Aşlamacı
PMID: 18400644  Pages 171 - 173
Abstract |Full Text PDF

20.The experience with the Epiclose®-T vascular access closure device: a human study
Soner Sanioğlu
PMID: 18400645  Page 174
Abstract |Full Text PDF

21.Can a city population represent a whole country? Essentials of study design for epidemiologic studies/Serum lipid profiles including non-high density lipoprotein cholesterol levels in Turkish school-children
Mehmet Yokuşoğlu, Mehmet Uzun, Oben Baysan
PMID: 18400646  Pages 174 - 175
Abstract |Full Text PDF

22.An isolated case of left ventricular non-compaction with sick sinus syndrome
Oben Baysan, Cem Köz, Mehmet Uzun, Mehmet Yokuşoğlu
PMID: 18400647  Pages 175 - 176
Abstract |Full Text PDF

23.Giant paraanastomotic iliac artery pseudoaneurysm
Bilgin Emrecan, İbrahim Gökşin
PMID: 18400649  Page 177
Abstract |Full Text PDF

24.Posttraumatic pneumopericardium and bilateral pneumothorax
Rasih Yazkan, Berkant Özpolat, Ali Cemal Düzgün, Süheyla Doğan
PMID: 18400648  Pages 177 - 178
Abstract |Full Text PDF

25.In memoriam of Prof. Dr. Baki Kamsuoğlu
Esmeray Acartürk
Page 179
Abstract |Full Text PDF

26.Our painful loss: Professor Dr. Zulpukar Kudaiberdiev (1939-2008)

Page 180
Abstract |Full Text PDF

27.Percutaneous coronary intervention performed for the revascularization of a single coronary artery originating from the right sinus of Valsalva
Mutlu Vural, Bayram Bağırtan, Öcal Karabay
PMID: 18400623  Page E8
Abstract |Full Text PDF

28.Single coronary artery diagnosed by multidedector computed tomography
Cihan Duran, Demet Erciyes, Mustafa Şirvancı, Murat Şener, Murat Gülbaran
PMID: 18400625  Page E9
Abstract |Full Text PDF

29.Coronary artery evaluation in Kawasaki disease by dual source multi-detector CT coronary angiography in children
Serkan Gelmez, Arda Saygılı, Ercan Tutar, Muzaffer Olcay Çizmeli
PMID: 18400624  Pages E9 - E10
Abstract |Full Text PDF

30.An interesting case of coronary artery fistula: diagonal artery-, left circumflex artery-and right coronary artery to left ventricle fistulas
Yasin Türker, Selahattin Akçay, Mehmet Özaydın
PMID: 18400616  Pages E10 - E11
Abstract |Full Text PDF

31.A case of coronary artery aneurysm resulted in sudden death
Şenay Funda Bıyıkoğlu, Sarper Ökten, Ayça Boyacı, Hatice Selçuk, Erdal Duru
PMID: 18400617  Pages E11 - E12
Abstract |Full Text PDF

32.Thrombotic lesion of saphenous vein graft resolved by antiaggregant therapy
Abdi Bozkurt, Abdurrahman Tasal, Esmeray Acartürk
PMID: 18400618  Page E12
Abstract |Full Text PDF

33.The accordion phenomenon
Sedat Türkoğlu, Timur kaynak
PMID: 18400619  Page E12
Abstract |Full Text PDF

34.A rare radiodiagnostic finding: totally and severely calcified aorta-the Porcelain aorta
Serkan Çay
PMID: 18400620  Page E13
Abstract |Full Text PDF

35.The imaging of the aortic fibrous membrane associated with Williams syndrome
Hacı Çiftçi, Özcan Özeke, Fikri Kutlay, Uğur Yuvanç
PMID: 18400621  Page E13
Abstract |Full Text PDF

36.Left pulmonary artery thrombosis diagnosed by transesophageal echocardiography
Hamza Duygu, Filiz Özerkan, Mustafa Akın
PMID: 18400622  Page E14
Abstract |Full Text PDF

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